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Identificação

Identificação pessoal

Nome completo
Ana Filipa Carvalho Marques Dias

Identificadores de autor

Ciência ID
B213-35E0-CF55
Formação
Grau Classificação
2018/03 - 2018/04
Concluído
Laboratory Animal Science Course for Function A (former FELASA Category B) for use of Zebrafish (Outros)
Instituto de Medicina Molecular, Portugal

Universidade de Lisboa Faculdade de Medicina, Portugal
2013/03 - 2017/10
Concluído
Biomedical Sciences (Doutoramento)
Universidade do Porto Instituto de Ciências Biomédicas Abel Salazar, Portugal
"The peroxisomal matrix protein import machinery – a PEX5-centered perspective" (TESE/DISSERTAÇÃO)
2009/09 - 2011/11
Concluído
Molecular Genetics and Biomedicine (Mestrado)
Universidade Nova de Lisboa Faculdade de Ciências e Tecnologia, Portugal
18
2006/09 - 2009/07
Concluído
Biology - Molecular Biology and Genetics with Minor in Biochemistry (Licenciatura)
Universidade de Lisboa Faculdade de Ciências, Portugal
17
Projetos

Bolsa

Designação Financiadores
2020/12/01 - 2022/03/31 Senescent cells and their secretory phenotype: novel targets for spinal cord repair
MC-36-2020
Investigador Pós-doutorado
Instituto de Medicina Molecular, Portugal
Santa Casa da Misericórdia de Lisboa
2020/01/08 - 2022/03/31 The function of induced-senescent cells in spinal cord regeneration
PTDC/MED-NEU/30428/2017
Investigador Pós-doutorado
Instituto de Medicina Molecular, Portugal
Fundação para a Ciência e a Tecnologia
2018/02/01 - 2020/01/07 Consortium for Genetically Tractable Organisms - CONGENTO
LISBOA-01-0145-FEDER-022170
Bolseiro de Pós-Doutoramento
Instituto de Medicina Molecular, Portugal
2017/11/01 - 2018/01/31 Porto Neurosciences and Neurologic Disease Research Initiative at i3S
NORTE-01-0145-FEDER-000008
Bolseiro de Pós-Doutoramento
Universidade do Porto Instituto de Biologia Molecular e Celular, Portugal

Universidade do Porto Instituto de Investigação e Inovação em Saúde, Portugal
2013/03/01 - 2017/02/28 The peroxisomal matrix protein import machinery - a PEX5-centered perspective
SFRH/BD/87352/2012
Bolseiro de Doutoramento
Universidade do Porto Instituto de Biologia Molecular e Celular, Portugal

Universidade do Porto Instituto de Investigação e Inovação em Saúde, Portugal
Fundação para a Ciência e a Tecnologia
2012/02/01 - 2012/12/31 The crosstalk between lipid antigen presentation and the pathogenic mechanisms of Lysosomal Storage Diseases
PTDC/SAU-ORG/110112/2009
Bolseiro de Investigação
Universidade do Porto Instituto de Biologia Molecular e Celular, Portugal
Produções

Publicações

Artigo em revista
  1. "Mouse Spinal Cord Vascular Transcriptome Analysis Identifies CD9 and MYLIP as Injury-Induced Players.". International journal of molecular sciences (2023): https://europepmc.org/articles/PMC10094762.
    10.3390/ijms24076433
  2. Paramos-de-Carvalho, Diogo; Martins, Isaura; Cristóvão, AM; Dias, Ana Filipa; Neves-Silva, Dalila; Pereira, T; Chapela, D; et al. "Targeting senescent cells improves functional recovery after spinal cord injury.". Cell reports (2021): https://doi.org/10.1016/j.celrep.2021.109334.
    10.1016/j.celrep.2021.109334
  3. Diogo Paramos-de-Carvalho; Isaura Martins; Ana Margarida Cristóvão; Dias, Ana Filipa; Dalila Neves-Silva; Telmo Pereira; Diana Chapela; et al. "Depletion of senescent cells improves functional recovery after spinal cord injury". bioRxiv (2020):
    https://doi.org/10.1101/2020.10.28.358762
  4. Dias, Ana. "Lipid Antigen Presentation by CD1b and CD1d in Lysosomal Storage Disease Patients.". Frontiers in immunology (2019):
    10.3389/fimmu.2019.01264
  5. Pedrosa, AG; Francisco, T; Bicho, D; Dias, Ana; Barros-Barbosa, A; Hagmann, V ; Dodt, G; Rodrigues, TA; Azevedo, JE. "Peroxisomal monoubiquitinated PEX5 interacts with the AAA ATPases PEX1 and PEX6 and is unfolded during its dislocation into the cytosol.". The Journal of biological chemistry (2018): http://europepmc.org/articles/PMC6065197.
    10.1074/jbc.RA118.003669
  6. Francisco T; Rodrigues TA; Dias AF; Barros-Barbosa A; Bicho D; Azevedo JE. "Protein transport into peroxisomes: Knowns and unknowns.". BioEssays : news and reviews in molecular, cellular and developmental biology (2017): http://europepmc.org/abstract/med/28787099.
    10.1002/bies.201700047
  7. Dias AF; Rodrigues TA; Pedrosa AG; Barros-Barbosa A; Francisco T; Azevedo JE. "The peroxisomal matrix protein translocon is a large cavity-forming protein assembly into which PEX5 protein enters to release its cargo.". The Journal of biological chemistry (2017): http://europepmc.org/abstract/med/28765278.
    10.1074/jbc.m117.805044
  8. Francisco T; Dias AF; Pedrosa AG; Grou CP; Rodrigues TA; Azevedo JE. "Determining the Topology of Peroxisomal Proteins Using Protease Protection Assays.". Methods in molecular biology (Clifton, N.J.) (2017): http://europepmc.org/abstract/med/28409448.
    10.1007/978-1-4939-6937-1_3
  9. Rodrigues TA; Francisco T; Dias AF; Pedrosa AG; Grou CP; Azevedo JE. "A cell-free organelle-based in vitro system for studying the peroxisomal protein import machinery.". Nature protocols (2016): http://europepmc.org/abstract/med/27831570.
    10.1038/nprot.2016.147
  10. Dias AF; Francisco T; Rodrigues TA; Grou CP; Azevedo JE. "The first minutes in the life of a peroxisomal matrix protein.". Biochimica et biophysica acta (2016): http://europepmc.org/abstract/med/26408939.
    10.1016/j.bbamcr.2015.09.025
  11. Pereira CS; Azevedo O; Maia ML; Dias AF; Sa-Miranda C; Macedo MF. "Invariant natural killer T cells are phenotypically and functionally altered in Fabry disease.". Molecular genetics and metabolism (2013): http://europepmc.org/abstract/med/23433711.
    10.1016/j.ymgme.2013.01.018