Profesor Titular (area of Physiology) since 1989. Since then, his research has been directed to the field of inflammation in aspects such as its relationship with aging, the effects of oxygen free radicals, production of extracellular matrix and fibrosis, pain and renal diseases. Committed to research because it keeps him updated as a teacher and it allows him to train Ph. D. students and to attract Ph. Ds to the University. His scientific career in recent years has focused on research in retinoids, prostaglandin E2 and its receptors, always within the context of inflammation and its treatment. The result of these studies was the confirmation of the role of COX / PGE2 in the mechanism of therapeutic action of retinoic acids in different in vivo and in vitro models of renal injury. More recently, he has focused on PGE2 itself as a key regulator with unsuspected functions and confirmed its role in the regulation of the expression of transcription factors such as HIF-1a and RARß, on which determined effects of hypoxia, cisplatin, retinoids and inflammatory cytokines. Not only in renal cells, but also prostate cells. Currently, the medium-term scientific-technical objective of its line of research is to explore the physiological and pathological functions of intracellular PGE2 in the context of acute kidney injury, renal fibrosis and prostatic proliferative pathology, as well as diagnostic and therapeutic applications of this knowledge. We are talking about a practically unexplored field, in which there are only a dozen previous works on the subject (including ours). We think that it is due to the novelty of the concept that PGE2 regulates functions acting on intracellular receptors that published works have not received as much interest as we believe that sooner or later they will wake up as pioneers in this field.